Understanding the fundamental processes that drive athletic performance and the revolutionary approach to optimizing recovery.
The immense power of the equine athlete is generated within the sarcomere, the fundamental engine of muscle. The contraction process, known as the Sliding Filament Theory, is an intricate electrochemical event where nerve impulses trigger the release of calcium ions, allowing the proteins actin and myosin to bind and pull, shortening the sarcomere and generating force.
Nerve impulses trigger the release of neurotransmitters at the neuromuscular junction.
Calcium ions are released from the sarcoplasmic reticulum into the muscle fiber.
Actin and myosin proteins bind together, forming cross-bridges.
The sarcomere shortens, generating the immense force needed for athletic performance.
Strenuous exercise, whether from the explosive power of a racehorse or the sustained effort of a human athlete, causes microscopic damage to the fundamental engines of muscle: the sarcomeres.
As this animation shows, the initial impact of strain is a physical disruption. The elegant, highly organized structure of healthy sarcomeres implodes into dysfunction. This mechanical damage triggers a powerful inflammatory response that can lead to neutrophil recruitment, the release of pro-inflammatory mediators, and the stimulation of pain receptors (nociceptors), causing the deep, aching soreness that directly impacts the next day's performance.
Inflammatory Management – The device mobilizes neutrophils, macrophages, and platelets to address tissue stress before it progresses to injury. For elite athletes like Thoroughbreds, Quarter Horses, and Grand Prix jumpers, this proactive management of tissue stress is critical. Their stoic nature often masks the micro-damage from competition, and by the time clinical signs appear, significant underlying dysfunction may already be present. The K2 allows caretakers to move from reactive treatment to proactive conditioning.
Myofibril disruption from intense training
Neutrophil recruitment and mediator release
Nociceptor stimulation affecting performance
The K2 device's Directed Cellular Mobility Current (EDCMC) technology targets the complex network of muscles that power equine movement. From the superficial muscles visible on the surface to the deep stabilizing muscles along the spine, the K2 provides comprehensive treatment for optimal performance and recovery.
For decades, equine therapy has focused on passive support—managing symptoms while waiting for the body's own slow healing cascade. The K2 represents a paradigm shift to active, directed regeneration. By restoring the horse's natural bioelectric signals and using galvanotaxis to guide cellular migration, the K2 actively orchestrates the repair process, leading to a faster, stronger, and more organized recovery.
Discover how our revolutionary approach to cellular healing can transform your equine care practice.